Dab tsi yog cov tshuaj tiv thaiv?

Cov txheej txheem inflammatory yog, feem ntau, qhov lus teb ntawm lub cev los ua ib qho kev sim ua kom paub txog cov kab xev fais fab kis mus rau hauv nws. Yog li, qhov ua kom pom tseeb ntawm qhov lesion yog qhov tsawg thiab tus neeg muaj mob kis puas raug mob. Tab sis lub cev tsis tas yuav muaj peev xwm tiv nrog tus kab mob. Txhawm rau tiv thaiv kev puas tsuaj loj rau lub cev thiab cov nqaij, thaum lawv cov kev ua haujlwm yuav raug cuam tshuam, cov tshuaj tiv thaiv kab mob feem ntau siv. Yog tsis muaj lawv, koj tuaj yeem ua tau yam tsis muaj txiaj ntsim ntawm kev kho mob etiotropic. Yog tias peb tab tom tham txog cov kev mob ntawm cov kab mob hauv cov kab mob sib kis, thiab muaj kev puas tsuaj ntawm tus neeg mob, ces siv cov tshuaj tiv thaiv kom sai li sai tau yog qhov tsim nyog.

Tag nrho cov tshuaj tua kab mob tau muab faib ua peb pawg ua ntej: steroid, non-steroidal thiab qeeb acting.

Steroids

Cov no muaj xws corticosteroids. Keeb kwm nws yog: cortisone thiab hydrocortisone, tau los ntawm lub supragens. Tam sim no ib tug xov tooj ntawm cov tshuaj tau ntxiv nrog cov khoom siv hluavtaws: prednisolone, methylprednisolone, fluorinated derivatives - dexamethasone, triamcinolone, flumethasone, betamethasone. Steroids khwb khuav inhibit A2 phospholipase, thiab muaj kev cuam tshuam cov nyhuv. Cov kev qhia rau kev siv tshuaj steroids yog txhua hom mob rheumatism. Kev kho mob ntev, ntev txog 2 lub hlis, feem ntau nrog cov tshuaj uas tsis yog steroid.

Txhais tau tias kev ua qeeb

Cov antiinflammatory ntsiav tshuaj siv los kho mauj txhab ntawm connective cov ntaub so ntswg ntawm rheumatoid mob caj dab. Cov no yog cov tshuaj ntawm kev kho mob yooj yim nrog kev qeeb qeeb, uas ua rau nws tus kheej tshwm sim hauv ob peb lub hlis. Cov no muaj xws li kub, hingamine (delagil, chloroquine), penicillinamine, cytostatics, thiab lwm tus.

Cov tshuaj non-steroidal anti-inflammatory (NSAIDs)

Cov pab pawg no yog cov feem ntau. Nonsteroidal anti-inflammatory ntsiav tshuaj kuj muaj antipyretic thiab analgesic kev txiav txim. Muaj ntau yam kev ua ub no thiab kev ua haujlwm siab muaj txiaj ntsig rau lawv nrog cov chaw zoo. Txhua txhua hnub, kev npaj ntawm pawg neeg no tau txais los ntawm ntau tshaj li 30 lab tus neeg nyob thoob ntiaj teb, muaj li ib nrab ntawm lawv muaj hnub nyoog 60 xyoo. Ntau tus neeg yuav cov tshuaj hauv cov chaw muag tshuaj tsis tas yuav ntawv nqa tshuaj.

Cov NSAIDs tau muab faib ua pawg, nyob ntawm seb cov qauv tshuaj thiab qhov xwm txheej ntawm qhov ua. Thawj yog xws li cov tshuaj tiv thaiv kab mob siab ntawm kev ua haujlwm siab. Qhov no salicylates (nrog rau tag nrho lub npe hu aspirin), pyrazolidine (phenylbutazone), indoleacetic derivatives (indomethacin, sulindac) thiab phenylacetic (diclofenac) acids, oxicams (piroxicam thiab lwm tus neeg.), Derivatives ntawm propionic acid (ibuprofen ketaprofen, naproxen, etc. .). Rau tib pab pawg xws li qee cov non-acidic derivatives, piv txwv li, alkanones (nanobeton), sulfonamide derivatives (nimesulide, rofecoxib).

Kev npaj ntawm lub thib ob pab pawg neeg muaj ib tug tsis muaj zog anti-inflammatory kev ua si. Los ntawm txoj kev, lawv muaj xws li nrov paracetamol.

Cov nyhuv ntawm cov NSAIDs yog ua raws li qhov kev cuam tshuam ntawm cyclooxygenase (COX), ib qho enzyme muab kev koom tes hauv cov khoom cua ntawm prostaglandins. Cov tom kawg yog cov tswv cuab ntawm cov txheej txheem inflammatory, tsim cov tsos mob ntawm tus mob syndrome thiab kub kub hloov (kub cev).

Cov NSAIDs muaj ntau yam tshwm sim, tiam sis tsis ntev los no cov tshuaj yeeb dej cawv nyuam qhuav pib tshwm sim (meloxicam, tenoxicam, nabumetone, solpaflex), uas yog xyuam xim tsis zoo rau cov mob prostaglandins, txo cov kev tsis haum. Paub txog ntau cov kev mob tshwm sim loj heev uas ntau yam tshuaj tua kab mob yog txhaum, cov tshuaj tiv thaiv kab mob feem ntau nyiam yog tias muaj kev xaiv.