Vascular kab mob

Vascular kab mob raug muab faib ua tej pawg neeg.

Inflammatory dab.

Qhov no pab pawg neeg no muaj xws li tus thawj kws (mauj kev tsis haum) thiab theem nrab vasculitis. Pawg no kuj muaj xws li xws kab mob raws li vascular aortitis, phlebitis thiab thrombophlebitis.

Atherosclerosis.

Qhov no muaj xws li qeb txhab nyob rau hauv cov hlab ntsha ntawm ntau yam localization (hlwb, plawv, raum).

Embolism, thrombosis thiab thromboembolism yog nce "kho" thiab "phais" pathologies.

Tsis tas li ntawd vascular cov kab mob xws li ischemia ntawm ntau hom (xws li, cov lwm yam, nqua), endarteritis obliterans, ntshav qab zib macroangiopathy, predgangrenu thiab lwm tus neeg.

Raws li cov classical lub cev sawv ces dag thiab sib sib zog nqus venous system. Kev sib txuas lus nruab nrab ntawm lawv yog nqa tawm los ntawm txoj kev nyias-walled cov hlab ntsha (perforating leeg). Lawv tsis muaj ib tug tej yam nyob rau hauv lub tsim ntawm venous insufficiency. Lub ntsiab feature ntawm no tshuab yog muaj ib tug vascular valve system muab ib tug unidirectional txaus.

Tsis ntev los no, ntau npaum li cas mloog yog them mus rau lub venous kab mob. Nyob rau hauv kev, loj tseem ceeb yog txuas mus rau nws caj ces xwm. Nyob rau hauv Feem ntau, pub kis tsis tau tus kab mob, tab sis tsuas raws roj ntsha anomaly qauv ntawm cov hlab ntsha. Congenital tsis muaj peev xwm yuav manifested nrog tsis txaus li qub los yog cov khoom nyob rau hauv daim ntawv ntawm lawv cov anatomical underdevelopment. Thab plaub yam tseem ceeb rau cov tsim ntawm pathological kev hloov ntawm cov ntaub ntawv no yog hormonal ntshawv siab, lub cev overload.

Feem ntau ces pathologies muaj xws li:

- sai tau cov kev xav ntawm qaug zog nyob rau hauv lub ob txhais ceg tom qab qoj ib ce;

- xws li loog loog los yog tingling;

- tas mus li o ntawm ko taw;

- ib tug ntev lub sij hawm uas tsis yog-kho ceg ulcers.

Raws li ib tug tshwm sim ntawm capillary muaj nuj nqi mob kuj tsim venous kab mob. Vim hais tias lub siab nyob rau ntawd yog hloov me me txog ntsha phab ntsa. Hlab ntsha ua convex thiab o. Yog li, ib tug vascular daim phiaj ob txhais ceg.

Cov ua ntawm no pathological mob ntau. Lub ntsiab zoo tshaj provoking kws txawj hu rau ntev li hom rau ob txhais ceg.

Nyob rau hauv tas li ntawd, lub capillary muaj nuj nqi tsis meej ua rau kev noj cov zaub mov, mob, siab thiab cov hnyuv, ntxaug nyob rau hauv cov ntshav ncig. Thaum lub sij hawm cev xeeb tub, nws yog ib qho tseem ceeb mus saib koj qhov ceeb thawj, txwv tsis pub qhov tshaj qhov hnyav kuj yuav ua txoj kev loj hlob ntawm pathology.

Kuj muaj ntau tus neeg tsim lawv tus kheej cov teeb meem, quav dej quav cawv, haus luam yeeb, hnub raug, hormonal tshuaj.

Nws yuav tsum tau muab sau tseg tias cov tsos ntawm vascular mesh yog raug tsis tau tsuas yog rau qhov qis dua nqua. Raws li ib tug tshwm sim ntawm lub plawv mob muaj peev xwm tsim rosacea. Vascular net ntawm lub ntsej muag tshwm sim nyob rau hauv cov neeg uas muaj muaj zog tawv nqaij rhiab heev. Hais tias lawv muaj ntau susceptible mus rau lwm cov kub hloov mus hloov los, raws li zoo raws li lub phiv los ntawm tej yam kom hniav zoo nkauj khoom.

Tus thawj cov tsos mob ntawm rosacea yog recurrent burning thiab khaus. Tom qab ntawd voos npaj, feem ntau yog nyob rau hauv lub hauv pliaj, qhov ntswg thiab lub puab tsaig. Nyob rau hauv lub chav kawm ntawm tus kab mob no cov tsos mob ua mob hnyav dua thiab tshwm sim ntau ntau zaus. Nyob rau hauv lub tom ntej no theem ntawm tus kab mob muaj khaus liab ntawm daim tawv nqaij thiab tsim ntawm kab laug sab leeg.

Thaum ib tug thaum ntxov tej yam tshwm sim ntawm tus kab mob nyob rau hauv thiaj li yuav tiv thaiv kom txhob undesirable txim tsim nyog mus thov mus rau lub koom neeg.