Txhais tau hais tias "kokarboksilazu". Cov lus qhia rau kev siv

Kev npaj "kokarboksilazu" yog muaj nyob rau hauv cov hmoov daim ntawv no rau kev npaj ntawm txhaj tshuaj. Tus neeg saib xyuas belongs rau cov pab pawg neeg ntawm cov vitamins B (B1 ua ke nrog B12 thiab B6). Cov tshuaj tom qab thawj coj parenterally undergoes sai thiab ua kom tiav haum. Lub medicament yuav txeem mus rau hauv tag nrho cov ntaub so ntswg, tab sis nws siab tshaj plaws concentration pom nyob rau hauv lub plawv, ob lub raum, lub hlwb. Thauj cov ntshav yog nqa tawm tsuas yog nrog rau cov ntshav liab. Cov metabolism yuav siv sij hawm qhov chaw nyob rau hauv lub siab lub hlwb. Feem yog nqa tawm nyob rau hauv ib tug unmodified daim ntawv. Tus nqi tas medicament nyob rau hauv lub koob tau txais. Qhov no feature tiv thaiv tsub zuj zuj ntawm cov tshuaj nyob rau hauv lub cev.

kev ua si mechanism

Txhais tau hais tias "carboxylase" (phau ntawv qhia no muaj cov ntaub ntawv xws li) txhawb kom cov ntaub so ntswg metabolism, nws muaj metabolic los. Cov tshuaj tsub kom tus haum qabzib, thiab pab txoj kev trophism nyob rau hauv lub paj hlwb cov ntaub so ntswg, thiab normalizes cov kev ua ntawm cov hlab plawv system.

Drug Administration "kokarboksilazu". Cov lus qhia. nqi

Tus nqi ntawm cov tshuaj nyob rau hauv cov khw - los ntawm 150 rubles. Txhais tau hais tias "kokarboksilazu" cov lus qhia rau kev siv pom zoo rau cov neeg mob nrog pathologies provoked teeb nyob rau hauv plab zom mov, parenteral kev noj haus los yog malabsorption. Los ntawm indications xws li Crohn tus kab mob, lub siab mob npuas paug, biliary system kab mob, mob raws plab, celiac kab mob. Prescribers thiab tom qab tshem tawm ntawm lub ileum. Pom zoo tshuaj rau metabolic mob, mob CAD flow, infarction, tsis ruaj tsis khov angina pectoris. Los ntawm indications kuj siv myocardial infarction. Thaum cov pathologies txhais tau tias "kokarboksilazu" phau ntawv qhia kom mus sau npe raws li ib tug ntxiv cov tshuaj. Kev kho mob yog cov tshuaj rau cov neeg mob uas lom los ntawm ethyl cawv thiab barbiturates, neuropathy, leeg mob, neuralgia. Los ntawm indications xws li perinatal hypoxic encephalopathy, mob ntsws, ua pa tsis ua hauj lwm. Tsa cov tshuaj kub taub hau, kub nyhiab, gipertireodizme, hemodialysis. Nyob rau hauv lub neej coj txoj kev kho neeg sawv cev pom zoo tom qab mob tus kab mob.

siv hom

Cov tshuaj ntawm cov tshuaj "kokarboksilazu" phau ntawv qhia xav kom koj nruab nyob rau hauv raws li cov mob ntawm tus neeg mob. Introduction ntawm cov tshuaj yog nqa tawm maj mam, tshaj kaum feeb. Ceev ceev txhaj yuav ua rau lub zos tshua. Nyob rau hauv kev, muaj peev xwm ua tau mob hnyav mob thiab ib tug burning nov ntawm nqaij tawv ntawm lub qhov koob. Ntau npaum rau cov neeg laus - tso dej los yog intramuscular txhaj ntawm 50-100 mg ib hnub ib zaug. Nyob rau hauv loj heev zaum (tiv thaiv ib tug tom qab ntawm dej cawv qaug cawv los yog ntshav qab zib coma) tshuaj yog tau tso cai rau rov qab mus tom qab 1-2 xuab moos ntau npaum rau txij nkawm txoj kev kho -. 50 mg ib zaug ib hnub twg. Cov me nyuam cov tshuaj "kokarboksilazu" phau ntawv qhia kom cov nram qab no koob tshuaj:

• 3 lub hlis - 25 mg;
• mus txog rau 7 xyoo - 25-50 mg;
• 18 xyoo - 50-100 mg.

Tus zaus ntawm tshuaj - 1 lub sij hawm ib hnub. Duration ntawm kev kho mob - los ntawm kaum tsib rau peb caug hnub.