Hom ntawm edema: qhov kev piav qhia thiab kev kho mob

Yuav ua li cas yog cov hom ntawm edema? Yuav ua li cas lawv txawv thiab dab tsi yog qhov vim li cas rau txoj kev loj hlob? Cov lus teb rau cov lus nug no yuav muaj nyob rau hauv no tsab xov xwm. Peb kuj qhia koj txog yuav ua li cas koj yuav tau tshem ntawm cov tsis kaj siab tshwm sim.

Cov ntaub ntawv hais txog lub o

Hom ntawm edema nyob ntawm seb lawv qhov chaw nyob thiab ua rau txoj kev loj hlob. Lub xeev no yog yus muaj los ntawm ntau ntau txuam nrog kua nyob rau hauv cov ntaub so ntswg extracellular qhov chaw thiab tib neeg nruab nrog cev. Feem ntau cov feem ntau, edema cai tom qab waking li, thiab nyob rau thaum xaus ntawm ib tug tibneeg hu tauj coob hnub.

hauj lwm edema

Hom ntawm edema yog txiav txim nyob ntawm qhov ua rau. Txawm li cas los, lawv yog cov tag nrho zoo ib yam li txhua lwm yam. Edematous cov ntaub so ntswg yog muaj zog los yog mob me me qhia o. Heev feem ntau, tom qab kev sib cuag nrog nws (e.g., ntiv tes siab) ib co kua nplaum nyob twj ywm rau ntawm daim tawv nqaij, uas tej zaum yuav ploj tom qab ib tug ob peb lub vib nas this, los yog tom qab ib tug ob peb teev.

Cov ua rau edema

O muaj peev xwm tshwm sim rau ntau yam yog vim li cas. Feem ntau cov feem ntau, xws li ib tug pathological mob raug rau lub sab nqua. Hom ntawm ceg edema yog nyob ntawm seb qhov ua rau. Cov no muaj xws li nram qab no:

• loj load rau hauv lub qis nqua;
• ntev li dej kom tsawg, xws li ua ntej yuav mus pw;
• tiaj ko taw;
• impaired metabolism;
• nquag hla ntawm ob txhais ceg nyob rau hauv ib tug zaum txoj hauj lwm;
• ntev zaum mos dhau los yog tsawg rooj;
• rog;
• phem heev matched nkawm khau (eg, siab heeled los yog tiaj tus nkawm khau, nrog ze shafted, thiab lwm yam);
• varices (tshwj xeeb tshaj yog feem ntau xws edema cai nyob rau hauv lub yav tsaus ntuj, thiab tom qab pw tsaug zog - zoo).

Nws yuav tsum tau muab sau tseg tias o nyob rau hauv ob txhais ceg tej zaum yuav tsim raws li ib tug tshwm sim ntawm kev ua txhaum ntawm cov ntshav txaus thiab hydrostatic siab nyob rau hauv lub veins.

lwm yam ua rau

Dhau li ntawm tus saum toj no yog vim li cas txoj kev loj hlob ntawm edema, lawv tshwm sim muaj peev xwm yuav txuam nrog:

• txhaum ntawm cov hlab plawv tus ua si;
• raum kab mob;
• plab hnyuv kab mob;
• mob ua pa kab mob;
• txhaum ntawm cov qog nyob rau hauv lub subcutaneous cov ntaub so ntswg thiab daim tawv nqaij;
• thyroid kab mob;
• mob thrombophlebitis thiab lwm yam.

hom ntawm edema

Lub ntsiab yam ntawm edema muaj xws li cov nram qab no:

• Generalized (los yog txhaws lub plawv thiab raum, endocrine thiab cachectic).
• Laus (inflammatory, tshuaj lom thiab kev tsis haum).

Xav txog tag nrho cov hom ntawm cov ntaub so ntswg edema nyob rau hauv ntau yam.

Plawv edema thiab kev kho mob

Lawv tshwm sim raws li ib tug tshwm sim ntawm lub plawv tsis ua hauj lwm. Nrog xws li ib tug pathology yog txo mob cov zis thiab tsub kom hypotension.

Ib tug tseem ceeb luag hauj lwm nyob rau hauv lub pathogenesis ntawm mob edema ua si venous tawg thiab venous stasis. Xeem disturbs qub metabolism hauv cov nqaij, uas ntxiv oxidized khoom ntawm metabolism, thiab npaj acidosis. Raws li ib tug tshwm sim ntawm cov qauv no yog ntxhov vascular intercellular cementing tshuaj thiab tsub kom nws permeability. Qhov no tshwm sim, ua ke nrog ib tug kub siab sodium provokes zog, cov nqaijrog thiab dej los ntawm cov kab noj hniav ntawm cov hlab ntsha nyob rau hauv lub interstitium. Tom qab ntawd edema.

Yuav ua li cas los kho qhov no pathology? mob edema, kev kho mob yuav tsum tau tswj kev txhim kho myocardial contractile ua hauj lwm los ntawm tau txais mob glycosides, raws li zoo raws li enhanced dej thiab Na feem ntawm lub cev los ntawm diuretics.

Raum o thiab Solutions

Ua ntawm lub xeev no yog los txo cov oncotic siab nyob rau hauv cov ntshav ntshav. Qhov no tus txheej txheem yog vim proteinuria, uas yog cai nyob rau hauv qhov txhab ntawm lub glomerular apparatus ntawm ob lub raum. Tom qab ib tug tseem ceeb nce nyob rau hauv lub pom ntawm cov dej los ntawm cov hlab ntsha thiab cov hlab ntsha nyob rau hauv lub interstitium, nws resorption nyob rau hauv cov leeg txo sharply. Yog li, cov kua accumulates nyob rau hauv cov ntaub so ntswg thiab edema. Yuav ua li cas yuav nws yuav tshem tawm?

Raum edema tawm hauv tsuas yog thaum tau txais diuretics. Txawm li cas los, yuav tsum paub hais tias cov sib ntaus tawm tsam no o yuav tsis ua rau kom cov lag luam ntawm lub cuam tshuam nrog cev. Yog li ntawd, tag nrho cov yam ntawm cov loj dag zog ntawm cov tshuaj los yog zoo dua siv rau direct kev kho mob ntawm lub raum kab mob.

Maranticheskie los yog cachectic edema thiab txoj kev kho

Xws li ib tug pathological mob muaj loj thiab ceev nrooj kab mob hauv lub cev, raws li zoo raws li huab tej yam kev mob, nrog rau phem hlav, mob raws plab, protein starvation, beriberi, thiab hais txog.

Lub ntsiab ua maranticheskih cachectic los yog edema yog ib tug tseem ceeb yuav txo tau nyob rau hauv lub synthesis ntawm soluble ntshav cov nqaijrog thiab cov kev loj hlob ntawm hypoproteinemia thiab yuav txo tau ntawm protein synthesis nyob rau hauv connective ntaub so ntswg. Tsis tas li ntawd rau qhov no pathological mob yog yus muaj los ntawm ib tug tseem ceeb nce nyob rau hauv capillary permeability rau electrolytes thiab dej, uas tom qab ntxiv nyob rau hauv lub interstices.

Yuav ua li cas mus kho cov hom ntawm edema? Kev kho mob maranticheskih los yog cachectic pathologies txoj kev kho thiaj li yuav mus rau loj kab mob uas yuav muaj tau teev saum toj no.

Endocrine o thiab cov hau kev ntawm kev kho mob

Ua ntawm lub xeev no yog ua rau kom cov hydrophilic xwm ntawm protein lug ntawm nqaij thiab daim tawv nqaij. Heev feem ntau, qhov no pathology tshwm sim thaum myxedema (ie hypothyroidism). Rau ib tug kab mob yus muaj los ntawm cov protein ntau catabolism, raws li zoo raws li ib tug distortion ntawm lawv synthesis. Raws li ib tug tshwm sim ntawm txoj kev no ib tug lossis loj npaum li cas ntawm mucin yog tsim, i.e. ib hydrophilic colloid, uas yog tsim nyob rau hauv cov nqaij thiab daim tawv nqaij, uas ua rau ncua sij hawm nyob rau hauv cov dej thiab li o thiab kev loj hlob.

Yuav ua li cas yog lub o ntawm no hom? Yuav kom tshem tawm lawv, cov kws kho mob pom zoo kom siv cov thyroxine, piv txwv li cov thyroid hormones.

Tam sim no koj paub qhov yooj yim hom edema. Neeg uas lawv kov tsis tau, thiaj li generalized edema, uas tshwm sim vim internal malfunction nyob rau hauv lub cev. Yog hais tias lub o yog hnov mus rau lub liab qab qhov muag, nws raug xa mus rau ib tug laus o. Xav txog nws cov hom, yam ntxwv thiab cov hau kev ntawm kev kho mob tam sim no.

inflammatory edema

Yuav lub xeev no yuav preceded los ntawm ib tug kev tsis haum tshuaj? Edema vim qhov no zoo ntawm cov tshuaj tiv thaiv yog heev tiag tiag. Raws li cov ntaub ntawv ntawm cov kws txawj, inflammatory thiab tsis haum o tshwm sim vim lub zos disturbances nyob rau hauv capillary permeability. Tsis tas li ntawd, yog vim li cas rau qhov no tshwm sim tej zaum yuav ib tug tawg nyob rau hauv qhov kev tsom xam ntawm kev puas tsuaj.

Yuav kom tshem tawm tej lub xeev yuav tsum tshem tawm tej kev sib cuag nrog lub allergen, thiab kuj coj anti-inflammatory thiab antihistaminic agents.

tshuaj lom edema

Lub ntsiab yam ntawm pulmonary edema yog feem ntau tshwm sim los ntawm lub nqus tau pa tshuaj vapors lom txiav txim. Tsis tas li ntawd, ib tug pathological mob tej zaum yuav yog vim muaj kab tom thiab toxins ntawm cov kab mob. Nyob rau hauv tus ntawm cov tshuaj mas siv mam li nco dheev nce vascular permeability thiab cov nqaij mos metabolism yog ntxhov. Raws li ib tug tshwm sim, noog nyob rau hauv cov ntaub so ntswg mob Cheebtsam uas ua rau kom osmotic siab thiab o ntawm colloids. Nyob rau hauv ua ke nrog nrog ntau zog capillary permeability nws ua rau txoj kev loj hlob ntawm o.

Therapeutic ntsuas kom tshem tawm tshuaj lom edema yuav tsum tau tswj lub normalization ntawm metabolic dab nyob rau hauv cov ntaub so ntswg thiab cov stabilization ntawm capillary week.

Yuav kom saib lub ntsab lug

Raws li koj tau pom, muaj ntau ntau yam ntawm edema, uas yuav muaj kev sib txawv heev ua rau thiab yuav tsum tau sib txawv kev kho mob txoj kev. Tab sis dab tsi yuav tsis cuam tshuam rau tus neeg mob lub o nyob rau hauv lub cev, koj yuav tsum xub sab laj ib tug kws kho mob. Tom qab kev ntsuam xyuas ntawm tus neeg mob tus kws kho mob yog yuam ua hauj lwm mus nraim nws mus rau ib tug nqaim tshwj xeeb uas yuav paub meej tias tus mob thiab muab cov kev tsim nyog kho mob.

Peb tsis tau hais tias qhov kev ncua nyob rau hauv kev kho mob muaj peev xwm thab edema, thiab yog li ntawd nyuaj mob ntawm tus neeg. Tom qab ib tug generalized thiab laus o yuav txawm ua rau tuag.