Duodenal rwj

Duodenal rwj - ib tug ntau pathology. Tus kab mob cov neeg nyob rau niaj hnub lub zos yog sau npe ob zaug li yuav tshaj nyob deb nroog nyob, uas yog tej zaum vim cov loj loj puas hawv thiab lub siab lub ntsws tab kaum thiab kev nyuaj siab. duodenal rwj ntev ntawm ib tug neeg laus nce mus txog lub peb caug centimeters. Lub plab ua ke nrog rau daim siab thiab txiav occupies ib tug tseem ceeb txoj hauj lwm nyob rau hauv lub plab zom mov, lub synthesis ntawm digestive kua, lub cev muaj zog thiab kev khiav tawm muaj nuj nqi ntawm lub digestive ib ntsuj av.

Duodenal rwj: etiology

Zaum hais tias cov etiology ntawm tus kab mob yog txuam nrog huab tej yam tas li kuj nyob rau hauv tib neeg lub cev thiab provocative ua txhaum ntawm neurohumoral mechanisms ntawm kev cai ntawm ntau kabmob. Nws yuav tsum tau muab sau tseg tias kev nyuaj siab qhov teeb meem no kuj yuav txo qhov kuj tau tus kab mob. Perforated lub rwj ntawm lub duodenum yog feem ntau nrog los ntawm txoj kev loj hlob ntawm peritonitis. Qhov no pathology yog sau npe nyob rau hauv cov txiv neej ntau ntau. Raws li ib tug txoj cai, perforated ulcers nyob rau hauv lub caij nplooj ntoos hlav thiab lub caij nplooj zeeg lub sij hawm. Nyob rau hauv txoj kev loj hlob ntawm gastro ulcers tseem ceeb luag hauj lwm ua si los ntawm Helicobacter pylori Campylobacter. Sau pov thawj zaum pom tau hais tias 95 feem pua ntawm cov neeg ntawm duodenal ulcers tau tus kab mob Helicobacter pylori.

Duodenal rwj: pathogenesis

Kev nyuaj siab yam ua rau lub cev saw ntawm complex thiab sib xyaw dab. Cov tshua yog nrog kawg ntawm humoral thiab neural mechanisms regulating lub zog ntawm tag nrho cov lub cev, tshwj xeeb tshaj yog cov hnyuv thiab plab. Nyob rau hauv cov ntshav tsub kom lub concentration ntawm histamine thiab cov tshuaj hormones ntawm lub cortex ntawm lub adrenal caj pas, uas, nyob rau hauv lem, activates lub tso pa tawm ntawm pais plab kua txiv. Cov kua txiv tsub kom lub concentration ntawm free chloride proteolitichekih acid thiab enzyme (pepsin). Nyob rau tib lub sij hawm nws inhibited lub tso pa tawm thiab hnoos qeev viscosity, li no zuj zus lawm lub barrier muaj nuj nqi ntawm lub mucosa. Cov nqaij ntshiv ntawm cov hnyuv thiab plab spazmiruyutsya, nce capillary permeability, muaj degeneration ntawm ntaub so ntswg, uas ua rau yus txoj kev loj hlob ntawm inflammatory dab. Tom ntej no tsim erosions thiab ulcers nyob rau hauv lub pais plab mucosa thiab duodenal ulcers.

Duodenal rwj txuam nrog tas kawg ntawm lub plab, siab, pancreas, hnyuv, thiab lwm yam kabmob. Ib tug tseem ceeb ib feem ntawm lub pathogenesis - mismatch ua kom cov muaj peev xwm ntawm pais plab los yog plab hnyuv txheem thiab mucosa barrier muaj nuj nqi. Nrog kev puas tsuaj rau cov hlab ntsha tshwm sim nyob rau hauv los ntshav rwj cheeb tsam.

Rwj ntawm lub duodenum: cov kev kho mob ntawm pej xeem cov tshuaj

Zoo kho nyhuv nyob rau hauv cov kev kho mob ntawm duodenal ulcers kev cai yog tus nqi uas muaj li nram qab no Cheebtsam: plantain, ntau npaum chamomile, peppermint, Calendula, yarrow, St. John lub wort. Cov tshuaj ntsuab nyob rau hauv tus sau, anti-inflammatory, antispasmodic, choleretic, laxative thiab astringent kev txiav txim.